What benefits of Colletotrichum gloeosporioides have been cited in the scientific literature?
The literature identifies several agents reported to inhibit C. gloeosporioides growth: lipopeptide biosurfactants fengycin A and fengycin B from Bacillus velezensis PW192 suppress it starting at 0.5 mg of crude acid-precipitated extract; n-butanol extracts of Bacillus strain FZB42 produce a bacteriostatic zone of 21.74 mm; Trichoderma asperellum peptaibols achieve 92.2% in vitro growth inhibition; and Burkholderia vietnamiensis FBCC-B8049 suppresses the fungus via direct antagonism and volatile organic compounds in dual-culture and ex vivo pepper-fruit assays.
1Potential claims from the literature
Reported as a phytopathogen causing anthracnose disease on plants, with antifungal biosurfactants (lipopeptides fengycin A and fengycin B) from Bacillus velezensis PW192 shown to inhibit its growth starting from 0.5 mg of acid-precipitated crude biosurfactant
Reported as a foliar pathogen (Colletotrichum gloeosporioides aff var. yerbae) causing disease in yerba mate (Ilex paraguariensis) cultivation
Reported to be inhibited by the n-butanol extract of Bacillus strain FZB42, with a bacteriostatic circle diameter of 21.74 ± 0.40 mm
Reported to be inhibited by transgenic Pelargonium graveolens constitutively overexpressing the Thchit42 gene, which demonstrated antifungal activity against C. gloeosporioides
Reported to produce four undescribed plant growth inhibitory indole derivatives (colletotriauxins A–D) as well as indole-3-acetic acid (IAA) and indole-3-acetamide, with colletotriauxins inhibiting Lepidium sativum seedling growth more strongly than IAA
Reported to have its growth slightly inhibited by plantain (Musa paradisiaca) stem leachate in an agar well diffusion assay
Reported to be inhibited by peptaibol extracts from Trichoderma asperellum at a growth inhibition rate of 92.2% in vitro
Reported to be suppressed by Burkholderia vietnamiensis FBCC-B8049 in dual-culture assays and ex vivo assays on pepper fruits, with inhibition attributed to direct antagonism and volatile organic compounds (VOCs)
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